We report a case of an acute ST-segment elevation myocardial infarction in a patient with traditional cardiac risk elements using supraphysiologic doses of supplemental, intramuscular testosterone. In addition, this affected person additionally had polycythemia, likely secondary to high-dose testosterone. The patient underwent profitable percutaneous intervention of the right coronary artery. Physicians and patients should pay consideration to the scientific penalties of anabolic steroid abuse. Development of an atheromatous plaque perpetuates endothelial dysfunction and promotes platelet aggregation and intracoronary thrombus formation (Ajayi et al 1995; Nieminen et al 1996).
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- Unfortunately, the information on the use of high dose anabolic steroids in humans are primarily supplied as case stories or small studies that lack enough control teams.
- Patients commonly develop left ventricular hypertrophy, which may be associated to hypertension, one other opposed effect of AAS.
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Rhoden EL, Morgentaler A. Risks of testosterone-replacement therapy and recommendations for monitoring. Testosterone substitute in hypogonadal males with angina improves ischaemic threshold and quality www.camsoda.com of life. Ajayi AAL, Mathur R, Halushka PV. Testosterone improve human platelet thromboxane A2 receptor density and aggregation responses.
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Anabolic Steroids, Acute Myocardial Infarction And Polycythemia: A Case Report And Evaluation Of The Literature
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Erythrocytosis has been reported as a potential effect of testosterone, which can be helpful in sufferers with anemia. The biggest risk of erythrocytosis occurs with injectable types of testosterone in contrast with transdermal preparations, especially in supraphysiologic doses. An enhance of up to 44% of the baseline hematocrit has been reported .
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